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Tubeimoside-1, a triterpenoid saponin, induces cytoprotective autophagy in human breast cancer cells in vitro via Akt-mediated pathway

Early correction of energy metabolic reprogramming in TECs, along with the restoration of mitochondrial function, is considered a potentially valuable therapeutic approach to reduce the severity and long-term consequences of AKI ( This review outlines the metabolic transitions observed during AKI, emphasizing the underlying pathophysiological mechanisms and key regulatory pathways implicated in mitochondrial dysfunction and energy metabolism

At first, general blurriness may make it difficult to read in low light or read street signs

doi: 10.1186/s13046-021-02012-7 50 MaWQSunXJZhuYLiuNF